File:IPLab6ChronicRejection1IPLab6ChronicRejection1b.jpg|This is a low-power photomicrograph of the kidney from this case of chronic transplant rejection. Note the focal areas of hemorrhage and inflammatory cell infiltrate in this section. File:IPLab6ChronicRejection2.jpg|This is a higher-power photomicrograph of kidney containing a section of blood vessel that demonstrates a marked neointimal proliferative response (1). In this case the lumen of the artery is obliterated. Also note the cellular infiltrate in the interstitium of the kidney (2) and the paucity of tubules. File:IPLab6ChronicRejection3IPLab6ChronicRejection2b.jpg|This is a photomicrograph of kidney with a focal area of hemorrhage around a small blood vessel (left) congestion and congestion sclerosis of the glomeruli. Note that there is a marked loss of renal tubules throughout this section with replacement by fibrous connective tissue. Also note the cellularity of the glomeruli. File:IPLab6ChronicRejection4IPLab6ChronicRejection3b.jpg|This is another area of renal cortex similar to the previous image. Note the fibrosis (1) and loss of renal tubules throughout this section. Also note the focus of inflammatory cells (2) indicating that despite the chromic nature of this lesion, there is still ongoing active rejection and renal damage. File:IPLab6ChronicRejection5IPLab6ChronicRejection4b.jpg|This high-power photomicrograph of glomeruli from this kidney demonstrates congestion (1), demonstrating increased cellularity of glomeruli with mesangial expansion, (arrows). There is generalized loss of tubules and a glomerulus that is almost completely obliterated or sclerosed replacement by fibrosis in this section (21). File:IPLab6ChronicRejection6IPLab6ChronicRejection5b.jpg|This is a photomicrograph of rejected kidney with a focus of cellular infiltrate (left) infiltrates and a small artery with neointimal proliferation and stenosis (arrow).

File:IPLab6ChronicRejection9IPLab6ChronicRejection9b.jpg|This is a high-power photomicrograph demonstrates the characteristic manifestations of a damaged chronic antibody-mediated rejection. The glomerulus. Note shows inflammatory cells within the loss of normal capillary structureloops (glomeruliitis), the accumulation of mesangial expansion matrix, and duplication (or multilamination) of the infiltration of large mononuclear cellscapillary basement membrane (arrow).