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Prostatic growth is dependent upon testicular androgens. Estrogen therapy leads to inhibition of pituitary gonadotropin and/or ACTH production which causes androgen deprivation. Newer drugs use specific testosterone receptor blocking mechanisms to regress prostate hyperplasia and prostate cancer.</spoiler>
* <spoiler text=" There appears to be an increase in the number of interstitial cells. Is there such a thing as Leydig cell hyperplasia?"> </spoiler>* <spoiler text=" "> No. The Leydig cells appear more prominent due to the paucity of germ cells.</spoiler>
{{IPLab 2}}
[[Category: IPLab:Lab 2]]