IPLab:Lab 10:Mucormycosis - Revision history

Peter Anderson: /* Clinical Summary */

2020-07-09T21:59:19Z

‎Clinical Summary

Peter Anderson: /* Clinical Summary */

2020-07-09T21:58:37Z

‎Clinical Summary

Peter Anderson: /* Autopsy Findings */

2020-07-09T21:57:46Z

‎Autopsy Findings

Seung Park at 16:35, 3 January 2014

2014-01-03T16:35:32Z

Seung Park: /* Images */

2013-08-30T01:45:45Z

‎Images

Seung Park: /* Images */

2013-08-24T03:34:28Z

‎Images

Seung Park: /* Images */

2013-08-24T03:34:18Z

‎Images

Seung Park: /* Journal Articles */

2013-08-24T03:32:04Z

‎Journal Articles

Seung Park: /* Reference */

2013-08-24T03:31:21Z

‎Reference

Seung Park at 04:24, 23 August 2013

2013-08-23T04:24:54Z

@@ Line 3: / Line 3: @@
 This 63-year-old white male was in his usual state of good health until eight weeks before his death when he developed sudden onset of shortness of breath. A thoracotomy was performed for plication of ruptured emphysematous blebs. Following improvement and discharge from the hospital he developed weakness, lethargy, and a left lower lobe lung infiltrate. The patient's condition soon deteriorated further, with almost every organ system having failed. The patient developed DIC and peripheral embolic phenomena, including gangrene of his extremities and face. A single antemortem blood culture grew Staphylococcus aureus.
-Autopsy revealed severe emphysema, severe widespread abscessiform and necrotizing pneumonia, and bacterial (Staphylococcus aureus) endocarditis of the pulmonic valve. The right internal carotid artery was occluded by a thrombus and there were areas of necrosis (due to CVAs) in the brain.
+Autopsy revealed severe emphysema, severe widespread abscessiform and necrotizing pneumonia, and bacterial (Staphylococcus aureus) endocarditis of the pulmonic valve. The right internal carotid artery was occluded by a thrombus and there were areas of necrosis due to CVAs (strokes) in the brain.
 == Images ==

@@ Line 1: / Line 1: @@
 == Clinical Summary ==
 This 63-year-old white male was in his usual state of good health until eight weeks before his death when he developed sudden onset of shortness of breath. A thoracotomy was performed for plication of ruptured emphysematous blebs. Following improvement and discharge from the hospital he developed weakness, lethargy, and a left lower lobe lung infiltrate. The patient's condition soon deteriorated further, with almost every organ system having failed. The patient developed DIC and peripheral embolic phenomena, including gangrene of his extremities and face. A single antemortem blood culture grew Staphylococcus aureus.
 == Images ==

← Older revision		Revision as of 21:57, 9 July 2020
Line 2:		Line 2:

	This 63-year-old white male was in his usual state of good health until eight weeks before his death when he developed sudden onset of shortness of breath. A thoracotomy was performed for plication of ruptured emphysematous blebs. Following improvement and discharge from the hospital he developed weakness, lethargy, and a left lower lobe lung infiltrate. The patient's condition soon deteriorated further, with almost every organ system having failed. The patient developed DIC and peripheral embolic phenomena, including gangrene of his extremities and face. A single antemortem blood culture grew Staphylococcus aureus.		This 63-year-old white male was in his usual state of good health until eight weeks before his death when he developed sudden onset of shortness of breath. A thoracotomy was performed for plication of ruptured emphysematous blebs. Following improvement and discharge from the hospital he developed weakness, lethargy, and a left lower lobe lung infiltrate. The patient's condition soon deteriorated further, with almost every organ system having failed. The patient developed DIC and peripheral embolic phenomena, including gangrene of his extremities and face. A single antemortem blood culture grew Staphylococcus aureus.
−
−	~~== Autopsy Findings ==~~
−
−	Autopsy revealed severe emphysema, severe widespread abscessiform and necrotizing pneumonia, and bacterial (Staphylococcus aureus) endocarditis of the pulmonic valve. The right internal carotid artery was occluded by a thrombus and there were areas of necrosis (due to CVAs) in the brain.

	== Images ==		== Images ==

@@ Line 18: / Line 18: @@
 File:IPLab10Mucor8.jpg|This is another high-power photomicrograph of the thrombus stained to outline the Mucor organisms (arrows).
 </gallery>
 == Study Questions ==

@@ Line 35: / Line 35: @@
 === Images ===
-* [http://peir.path.uab.edu/library/index.php?/tags/2158-mucormycosis PEIR Digital Library: Mucormycosis Images]
+* [{{SERVER}}/library/index.php?/tags/2158-mucormycosis PEIR Digital Library: Mucormycosis Images]
 * [http://library.med.utah.edu/WebPath/INFEHTML/INFECIDX.html WebPath: Infection]

@@ Line 32: / Line 32: @@
 === Journal Articles ===
+* Marcó del Pont J, De Cicco L, Gallo G, Llera J, De Santibanez E, D'agostino D.  [http://www.ncbi.nlm.nih.gov/pubmed/11429008 Hepatic arterial thrombosis due to Mucor species in a child following orthotopic liver transplantation].  ''Transpl Infect Dis'' 2000 Mar;2(1):33-5.
 === Images ===

@@ Line 28: / Line 28: @@
 == Additional Resources ==
 === Reference ===
+* [http://emedicine.medscape.com/article/222551-overview eMedicine Medical Library: Mucormycosis]
 === Journal Articles ===

@@ Line 25: / Line 25: @@
 The three primary sites of Mucor invasion are the nasal sinuses, lungs, and gastrointestinal tract.</spoiler>
 * <spoiler text="What special risks of Mucor infection are associated with diabetes?">In diabetics, the fungus may spread from nasal sinuses to the orbit, and subsequently the brain, giving rise to rhinocerebral mucormycosis.</spoiler>
 {{IPLab 10}}
 [[Category: IPLab:Lab 10]]