IPLab:Lab 1:Kidney Infarction - Revision history

Peter Anderson: /* Images */

2019-06-27T21:14:23Z

Images

← Older revision		Revision as of 16:14, 27 June 2019
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	== Images ==		== Images ==
	<gallery heights="250px" widths="250px">		<gallery heights="250px" widths="250px">
	File:~~IPLab1KidneyInfarction1~~.jpg\|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The ~~cut surface (right) shows several areas~~ of ~~infarction. The most recent~~ infarct is ~~seen at~~ the ~~top left~~ (arrow)~~. The surface~~ of the ~~kidney (left) shows a marked nodularity and roughening from scarring due to chronic hypertension.~~		File:IPLab1KidneyInfarction7.jpg\|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
	~~File:IPLab1KidneyInfarction2.jpg\|This~~ is ~~a closer view of the triangular-shaped infarction with the base at the cortical surface (1) and the apex~~ at the corticomedullary junction ~~(2)~~.
	File:IPLab1KidneyInfarction3.jpg\|This low-power photomicrograph of the kidney illustrates a sharply demarcated area of red discoloration extending from the capsule to the cortical medullary junction (arrow).		File:IPLab1KidneyInfarction3.jpg\|This low-power photomicrograph of the kidney illustrates a sharply demarcated area of red discoloration extending from the capsule to the cortical medullary junction (arrow).
	File:IPLab1KidneyInfarction4.jpg\|A higher-power photomicrograph shows the edge of this reddish area, illustrating coagulation necrosis (1) compared to the normal tissue (2). The necrotic ~~tubules~~ in this hemorrhagic, red infarct ~~are~~ hypereosinophilic. Compare the tubules on the right with the normal tubules seen in the left-hand portion of the slide. Note the interstitial hemorrhage which is associated with vascular leakage within this necrotic region in the tissue.		File:IPLab1KidneyInfarction4.jpg\|A higher-power photomicrograph shows the edge of this reddish area, illustrating coagulation necrosis (1) compared to the normal tissue (2). The necrotic tissue in this hemorrhagic, red infarct is hypereosinophilic. Compare the tubules on the right with the normal tubules seen in the left-hand portion of the slide. Note the interstitial hemorrhage which is associated with vascular leakage within this necrotic region in the tissue.
	File:IPLab1KidneyInfarction5.jpg\|This higher-power view of the infarct demonstrates retention of the tubular structure and cellular outlines. In the lower right-hand corner is a barely identifiable glomerulus (1). Note that, although the cellular architecture is retained, there are no nuclei within the renal tubular cells. The nuclei visible in this photomicrograph are the nuclei of inflammatory cells.		File:IPLab1KidneyInfarction5.jpg\|This higher-power view of the infarct demonstrates retention of the tubular structure and cellular outlines. In the lower right-hand corner is a barely identifiable glomerulus (1). Note that, although the cellular architecture is retained, there are no nuclei within the renal tubular cells. The nuclei visible in this photomicrograph are the nuclei of inflammatory cells.
	File:IPLab1KidneyInfarction6.jpg\|This section, taken at the cortical medullary junction, illustrates a blood vessel ~~in the upper right portion of the slide (arrow)~~ which is filled with thrombotic material. This vessel has occluded an end artery resulting in ischemia and infarction.		File:IPLab1KidneyInfarction6.jpg\|This section, taken at the cortical medullary junction, illustrates a blood vessel which is filled with thrombotic material (arrow). This vessel has occluded an end artery resulting in ischemia and infarction.
	File:IPLab1KidneyInfarction7.jpg\|This gross photograph of an infarcted kidney is from another case. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
	</gallery>		</gallery>

Peter Anderson: /* Autopsy Findings */

2019-06-27T21:06:02Z

Autopsy Findings

← Older revision		Revision as of 16:06, 27 June 2019
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	== Clinical Summary ==		== Clinical Summary ==
	This 48-year-old male died in a motor vehicle accident. An incidental finding at autopsy was a renal infarct which was reddish-tan in color, sharply delineated, and triangular in shape. The base of the infarct was located at the capsular surface and its apex at the corticomedullary junction.		This 48-year-old male died in a motor vehicle accident. An incidental finding at autopsy was a renal infarct which was reddish-tan in color, sharply delineated, and triangular in shape. The base of the infarct was located at the capsular surface and its apex at the corticomedullary junction.

	~~== Autopsy Findings ==~~
	An incidental finding at autopsy was a small renal lesion which was reddish-tan in color, sharply delineated, and triangular in shape. The base of the lesion was located at the capsular surface and its apex at the corticomedullary junction.

	== Images ==		== Images ==

Peter Anderson: /* Clinical Summary */

2019-06-27T21:05:12Z

Clinical Summary

← Older revision		Revision as of 16:05, 27 June 2019
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	== Clinical Summary ==		== Clinical Summary ==
	This 48-year-old ~~black~~ male ~~committed suicide by ingesting an unidentified toxin~~, ~~after which he went into profound shock~~ and ~~died~~.		This 48-year-old male died in a motor vehicle accident. An incidental finding at autopsy was a renal infarct which was reddish-tan in color, sharply delineated, and triangular in shape. The base of the infarct was located at the capsular surface and its apex at the corticomedullary junction.

	== Autopsy Findings ==		== Autopsy Findings ==

Peter Anderson: /* Virtual Microscopy */

2015-09-16T14:29:57Z

Virtual Microscopy

← Older revision		Revision as of 09:29, 16 September 2015
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	== Virtual Microscopy ==		== Virtual Microscopy ==
			=== Kidney Infarction ===
	<peir-vm>IPLab1KidneyInfarction</peir-vm>		<peir-vm>IPLab1KidneyInfarction</peir-vm>

			=== Normal Kidney ===
			<peir-vm>UAB-Histology-00114</peir-vm>

	== Study Questions ==		== Study Questions ==

Seung Park at 15:36, 3 January 2014

2014-01-03T15:36:32Z

← Older revision		Revision as of 10:36, 3 January 2014
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	File:IPLab1KidneyInfarction7.jpg\|This gross photograph of an infarcted kidney is from another case. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.		File:IPLab1KidneyInfarction7.jpg\|This gross photograph of an infarcted kidney is from another case. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
	</gallery>		</gallery>

			== Virtual Microscopy ==
			<peir-vm>IPLab1KidneyInfarction</peir-vm>

	== Study Questions ==		== Study Questions ==

Seung Park at 01:22, 30 August 2013

2013-08-30T01:22:20Z

← Older revision		Revision as of 20:22, 29 August 2013
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	=== Images ===		=== Images ===
	* [~~http://peir.path.uab.edu~~/library/index.php?/tags/52-kidney/30-infarct PEIR Digital Library: Kidney Infarct Images]		* [{{SERVER}}/library/index.php?/tags/52-kidney/30-infarct PEIR Digital Library: Kidney Infarct Images]
	* [http://library.med.utah.edu/WebPath/RENAHTML/RENALIDX.html WebPath: Renal Pathology Images]		* [http://library.med.utah.edu/WebPath/RENAHTML/RENALIDX.html WebPath: Renal Pathology Images]

Seung Park: /* Related IPLab Cases */

2013-08-21T06:10:52Z

Related IPLab Cases

← Older revision		Revision as of 01:10, 21 August 2013
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	* [[IPLab:Lab 5:Nodular Intercapillary Glomerulosclerosis\|Lab 5: Kidney: Nodular Intercapillary Glomerulosclerosis]]		* [[IPLab:Lab 5:Nodular Intercapillary Glomerulosclerosis\|Lab 5: Kidney: Nodular Intercapillary Glomerulosclerosis]]
	* [[IPLab:Lab 6:Glomerulonephritis\|Lab 6: Kidney: Glomerulonephritis]]		* [[IPLab:Lab 6:Glomerulonephritis\|Lab 6: Kidney: Glomerulonephritis]]
	* [[IPLab:Lab 6:Acute ~~Transplant~~ Rejection\|Lab 6: Kidney: Acute Transplant Rejection]]		* [[IPLab:Lab 6:Acute Rejection\|Lab 6: Kidney: Acute Transplant Rejection]]
	* [[IPLab:Lab 6:Chronic ~~Transplant~~ Rejection\|Lab 6: Kidney: Chronic Transplant Rejection]]		* [[IPLab:Lab 6:Chronic Rejection\|Lab 6: Kidney: Chronic Transplant Rejection]]

	{{Template:IPLab 1}}		{{Template:IPLab 1}}

	[[Category:IPLab:Lab 1]]		[[Category:IPLab:Lab 1]]

Seung Park at 23:31, 18 August 2013

2013-08-18T23:31:29Z

← Older revision		Revision as of 18:31, 18 August 2013
Line 6:		Line 6:

	== Images ==		== Images ==
	<gallery>		<gallery heights="250px" widths="250px">
	File:IPLab1KidneyInfarction1.jpg\|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The cut surface (right) shows several areas of infarction. The most recent infarct is seen at the top left (arrow). The surface of the kidney (left) shows a marked nodularity and roughening from scarring due to chronic hypertension.		File:IPLab1KidneyInfarction1.jpg\|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The cut surface (right) shows several areas of infarction. The most recent infarct is seen at the top left (arrow). The surface of the kidney (left) shows a marked nodularity and roughening from scarring due to chronic hypertension.
	File:IPLab1KidneyInfarction2.jpg\|This is a closer view of the triangular-shaped infarction with the base at the cortical surface (1) and the apex at the corticomedullary junction (2).		File:IPLab1KidneyInfarction2.jpg\|This is a closer view of the triangular-shaped infarction with the base at the cortical surface (1) and the apex at the corticomedullary junction (2).

Seung Park at 05:12, 16 August 2013

2013-08-16T05:12:36Z

← Older revision		Revision as of 00:12, 16 August 2013
Line 6:		Line 6:

	== Images ==		== Images ==
	~~{\| cellpadding="10"~~		<gallery>
	~~\| [[Image~~:IPLab1KidneyInfarction1.jpg~~\|frameless]]~~		File:IPLab1KidneyInfarction1.jpg\|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The cut surface (right) shows several areas of infarction. The most recent infarct is seen at the top left (arrow). The surface of the kidney (left) shows a marked nodularity and roughening from scarring due to chronic hypertension.
	\| This gross photograph shows a kidney that has been transected longitudinally at autopsy. The cut surface (right) shows several areas of infarction. The most recent infarct is seen at the top left (arrow). The surface of the kidney (left) shows a marked nodularity and roughening from scarring due to chronic hypertension.		File:IPLab1KidneyInfarction2.jpg\|This is a closer view of the triangular-shaped infarction with the base at the cortical surface (1) and the apex at the corticomedullary junction (2).
	\|-		File:IPLab1KidneyInfarction3.jpg\|This low-power photomicrograph of the kidney illustrates a sharply demarcated area of red discoloration extending from the capsule to the cortical medullary junction (arrow).
	~~\| [[Image~~:IPLab1KidneyInfarction2.jpg~~\|frameless]]~~		File:IPLab1KidneyInfarction4.jpg\|A higher-power photomicrograph shows the edge of this reddish area, illustrating coagulation necrosis (1) compared to the normal tissue (2). The necrotic tubules in this hemorrhagic, red infarct are hypereosinophilic. Compare the tubules on the right with the normal tubules seen in the left-hand portion of the slide. Note the interstitial hemorrhage which is associated with vascular leakage within this necrotic region in the tissue.
	\| This is a closer view of the triangular-shaped infarction with the base at the cortical surface (1) and the apex at the corticomedullary junction (2).		File:IPLab1KidneyInfarction5.jpg\|This higher-power view of the infarct demonstrates retention of the tubular structure and cellular outlines. In the lower right-hand corner is a barely identifiable glomerulus (1). Note that, although the cellular architecture is retained, there are no nuclei within the renal tubular cells. The nuclei visible in this photomicrograph are the nuclei of inflammatory cells.
	\|-		File:IPLab1KidneyInfarction6.jpg\|This section, taken at the cortical medullary junction, illustrates a blood vessel in the upper right portion of the slide (arrow) which is filled with thrombotic material. This vessel has occluded an end artery resulting in ischemia and infarction.
	~~\| [[Image~~:IPLab1KidneyInfarction3.jpg~~\|frameless]]~~		File:IPLab1KidneyInfarction7.jpg\|This gross photograph of an infarcted kidney is from another case. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
	\| This low-power photomicrograph of the kidney illustrates a sharply demarcated area of red discoloration extending from the capsule to the cortical medullary junction (arrow).		</gallery>
	\|-
	~~\| [[Image~~:IPLab1KidneyInfarction4.jpg~~\|frameless]]~~
	\| A higher-power photomicrograph shows the edge of this reddish area, illustrating coagulation necrosis (1) compared to the normal tissue (2). The necrotic tubules in this hemorrhagic, red infarct are hypereosinophilic. Compare the tubules on the right with the normal tubules seen in the left-hand portion of the slide. Note the interstitial hemorrhage which is associated with vascular leakage within this necrotic region in the tissue.
	\|-
	~~\| [[Image~~:IPLab1KidneyInfarction5.jpg~~\|frameless]]~~
	\| This higher-power view of the infarct demonstrates retention of the tubular structure and cellular outlines. In the lower right-hand corner is a barely identifiable glomerulus (1). Note that, although the cellular architecture is retained, there are no nuclei within the renal tubular cells. The nuclei visible in this photomicrograph are the nuclei of inflammatory cells.
	\|-
	~~\| [[Image~~:IPLab1KidneyInfarction6.jpg~~\|frameless]]~~
	\| This section, taken at the cortical medullary junction, illustrates a blood vessel in the upper right portion of the slide (arrow) which is filled with thrombotic material. This vessel has occluded an end artery resulting in ischemia and infarction.
	\|-
	~~\| [[Image~~:IPLab1KidneyInfarction7.jpg~~\|frameless]]~~
	\| This gross photograph of an infarcted kidney is from another case. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
	\|}

	== Study Questions ==		== Study Questions ==

Seung Park at 03:12, 16 August 2013

2013-08-16T03:12:34Z

← Older revision		Revision as of 22:12, 15 August 2013
Line 58:		Line 58:
	{{Template:IPLab 1}}		{{Template:IPLab 1}}

	[[Category:IPLab]]		[[Category:IPLab:Lab 1]]

← Older revision		Revision as of 16:14, 27 June 2019
Line 4:		Line 4:
	== Images ==		== Images ==
	<gallery heights="250px" widths="250px">		<gallery heights="250px" widths="250px">
	File:~~IPLab1KidneyInfarction1~~.jpg\|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The ~~cut surface (right) shows several areas~~ of ~~infarction. The most recent~~ infarct is ~~seen at~~ the ~~top left~~ (arrow)~~. The surface~~ of the ~~kidney (left) shows a marked nodularity and roughening from scarring due to chronic hypertension.~~		File:IPLab1KidneyInfarction7.jpg\|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
	~~File:IPLab1KidneyInfarction2.jpg\|This~~ is ~~a closer view of the triangular-shaped infarction with the base at the cortical surface (1) and the apex~~ at the corticomedullary junction ~~(2)~~.
	File:IPLab1KidneyInfarction3.jpg\|This low-power photomicrograph of the kidney illustrates a sharply demarcated area of red discoloration extending from the capsule to the cortical medullary junction (arrow).		File:IPLab1KidneyInfarction3.jpg\|This low-power photomicrograph of the kidney illustrates a sharply demarcated area of red discoloration extending from the capsule to the cortical medullary junction (arrow).
	File:IPLab1KidneyInfarction4.jpg\|A higher-power photomicrograph shows the edge of this reddish area, illustrating coagulation necrosis (1) compared to the normal tissue (2). The necrotic ~~tubules~~ in this hemorrhagic, red infarct ~~are~~ hypereosinophilic. Compare the tubules on the right with the normal tubules seen in the left-hand portion of the slide. Note the interstitial hemorrhage which is associated with vascular leakage within this necrotic region in the tissue.		File:IPLab1KidneyInfarction4.jpg\|A higher-power photomicrograph shows the edge of this reddish area, illustrating coagulation necrosis (1) compared to the normal tissue (2). The necrotic tissue in this hemorrhagic, red infarct is hypereosinophilic. Compare the tubules on the right with the normal tubules seen in the left-hand portion of the slide. Note the interstitial hemorrhage which is associated with vascular leakage within this necrotic region in the tissue.
	File:IPLab1KidneyInfarction5.jpg\|This higher-power view of the infarct demonstrates retention of the tubular structure and cellular outlines. In the lower right-hand corner is a barely identifiable glomerulus (1). Note that, although the cellular architecture is retained, there are no nuclei within the renal tubular cells. The nuclei visible in this photomicrograph are the nuclei of inflammatory cells.		File:IPLab1KidneyInfarction5.jpg\|This higher-power view of the infarct demonstrates retention of the tubular structure and cellular outlines. In the lower right-hand corner is a barely identifiable glomerulus (1). Note that, although the cellular architecture is retained, there are no nuclei within the renal tubular cells. The nuclei visible in this photomicrograph are the nuclei of inflammatory cells.
	File:IPLab1KidneyInfarction6.jpg\|This section, taken at the cortical medullary junction, illustrates a blood vessel ~~in the upper right portion of the slide (arrow)~~ which is filled with thrombotic material. This vessel has occluded an end artery resulting in ischemia and infarction.		File:IPLab1KidneyInfarction6.jpg\|This section, taken at the cortical medullary junction, illustrates a blood vessel which is filled with thrombotic material (arrow). This vessel has occluded an end artery resulting in ischemia and infarction.
	File:IPLab1KidneyInfarction7.jpg\|This gross photograph of an infarcted kidney is from another case. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
	</gallery>		</gallery>