IPLab:Lab 1:Kidney Infarction - Revision history

Peter Anderson: /* Images */

2019-06-27T21:14:23Z

‎Images

Peter Anderson: /* Autopsy Findings */

2019-06-27T21:06:02Z

‎Autopsy Findings

Peter Anderson: /* Clinical Summary */

2019-06-27T21:05:12Z

‎Clinical Summary

Peter Anderson: /* Virtual Microscopy */

2015-09-16T14:29:57Z

‎Virtual Microscopy

Seung Park at 15:36, 3 January 2014

2014-01-03T15:36:32Z

Seung Park at 01:22, 30 August 2013

2013-08-30T01:22:20Z

Seung Park: /* Related IPLab Cases */

2013-08-21T06:10:52Z

‎Related IPLab Cases

Seung Park at 23:31, 18 August 2013

2013-08-18T23:31:29Z

Seung Park at 05:12, 16 August 2013

2013-08-16T05:12:36Z

Seung Park at 03:12, 16 August 2013

2013-08-16T03:12:34Z

@@ Line 4: / Line 4: @@
 == Images ==
 <gallery heights="250px" widths="250px">
-File:IPLab1KidneyInfarction1.jpg|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The cut surface (right) shows several areas of infarction. The most recent infarct is seen at the top left (arrow). The surface of the kidney (left) shows a marked nodularity and roughening from scarring due to chronic hypertension.
+File:IPLab1KidneyInfarction7.jpg|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
 File:IPLab1KidneyInfarction3.jpg|This low-power photomicrograph of the kidney illustrates a sharply demarcated area of red discoloration extending from the capsule to the cortical medullary junction (arrow).
-File:IPLab1KidneyInfarction4.jpg|A higher-power photomicrograph shows the edge of this reddish area, illustrating coagulation necrosis (1) compared to the normal tissue (2). The necrotic tubules in this hemorrhagic, red infarct are hypereosinophilic. Compare the tubules on the right with the normal tubules seen in the left-hand portion of the slide. Note the interstitial hemorrhage which is associated with vascular leakage within this necrotic region in the tissue.
+File:IPLab1KidneyInfarction4.jpg|A higher-power photomicrograph shows the edge of this reddish area, illustrating coagulation necrosis (1) compared to the normal tissue (2). The necrotic tissue in this hemorrhagic, red infarct is hypereosinophilic. Compare the tubules on the right with the normal tubules seen in the left-hand portion of the slide. Note the interstitial hemorrhage which is associated with vascular leakage within this necrotic region in the tissue.
 File:IPLab1KidneyInfarction5.jpg|This higher-power view of the infarct demonstrates retention of the tubular structure and cellular outlines. In the lower right-hand corner is a barely identifiable glomerulus (1). Note that, although the cellular architecture is retained, there are no nuclei within the renal tubular cells. The nuclei visible in this photomicrograph are the nuclei of inflammatory cells.
-File:IPLab1KidneyInfarction6.jpg|This section, taken at the cortical medullary junction, illustrates a blood vessel in the upper right portion of the slide (arrow) which is filled with thrombotic material. This vessel has occluded an end artery resulting in ischemia and infarction.
+File:IPLab1KidneyInfarction6.jpg|This section, taken at the cortical medullary junction, illustrates a blood vessel  which is filled with thrombotic material (arrow). This vessel has occluded an end artery resulting in ischemia and infarction.
 </gallery>

@@ Line 1: / Line 1: @@
 == Clinical Summary ==
 This 48-year-old male died in a motor vehicle accident.  An incidental finding at autopsy was a renal infarct which was reddish-tan in color, sharply delineated, and triangular in shape. The base of the infarct was located at the capsular surface and its apex at the corticomedullary junction.
 == Images ==

@@ Line 1: / Line 1: @@
 == Clinical Summary ==
-This 48-year-old black male committed suicide by ingesting an unidentified toxin, after which he went into profound shock and died.
+This 48-year-old male died in a motor vehicle accident.  An incidental finding at autopsy was a renal infarct which was reddish-tan in color, sharply delineated, and triangular in shape. The base of the infarct was located at the capsular surface and its apex at the corticomedullary junction.
 == Autopsy Findings ==

@@ Line 17: / Line 17: @@
 == Virtual Microscopy ==
 <peir-vm>IPLab1KidneyInfarction</peir-vm>
 == Study Questions ==

@@ Line 15: / Line 15: @@
 File:IPLab1KidneyInfarction7.jpg|This gross photograph of an infarcted kidney is from another case. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
 </gallery>
 == Study Questions ==

@@ Line 33: / Line 33: @@
 === Images ===
-* [http://peir.path.uab.edu/library/index.php?/tags/52-kidney/30-infarct PEIR Digital Library: Kidney Infarct Images]
+* [{{SERVER}}/library/index.php?/tags/52-kidney/30-infarct PEIR Digital Library: Kidney Infarct Images]
 * [http://library.med.utah.edu/WebPath/RENAHTML/RENALIDX.html WebPath: Renal Pathology Images]

@@ Line 40: / Line 40: @@
 * [[IPLab:Lab 5:Nodular Intercapillary Glomerulosclerosis|Lab 5: Kidney: Nodular Intercapillary Glomerulosclerosis]]
 * [[IPLab:Lab 6:Glomerulonephritis|Lab 6: Kidney: Glomerulonephritis]]
-* [[IPLab:Lab 6:Acute Transplant Rejection|Lab 6: Kidney: Acute Transplant Rejection]]
+* [[IPLab:Lab 6:Acute Rejection|Lab 6: Kidney: Acute Transplant Rejection]]
-* [[IPLab:Lab 6:Chronic Transplant Rejection|Lab 6: Kidney: Chronic Transplant Rejection]]
+* [[IPLab:Lab 6:Chronic Rejection|Lab 6: Kidney: Chronic Transplant Rejection]]
 {{Template:IPLab 1}}
 [[Category:IPLab:Lab 1]]

@@ Line 6: / Line 6: @@
 == Images ==
-{| cellpadding="10"
+<gallery>
-| [[Image:IPLab1KidneyInfarction1.jpg|frameless]]
+File:IPLab1KidneyInfarction1.jpg|This gross photograph shows a kidney that has been transected longitudinally at autopsy. The cut surface (right) shows several areas of infarction. The most recent infarct is seen at the top left (arrow). The surface of the kidney (left) shows a marked nodularity and roughening from scarring due to chronic hypertension.
-| This gross photograph shows a kidney that has been transected longitudinally at autopsy. The cut surface (right) shows several areas of infarction. The most recent infarct is seen at the top left (arrow). The surface of the kidney (left) shows a marked nodularity and roughening from scarring due to chronic hypertension.
+File:IPLab1KidneyInfarction2.jpg|This is a closer view of the triangular-shaped infarction with the base at the cortical surface (1) and the apex at the corticomedullary junction (2).
-|-
+File:IPLab1KidneyInfarction3.jpg|This low-power photomicrograph of the kidney illustrates a sharply demarcated area of red discoloration extending from the capsule to the cortical medullary junction (arrow).
-| [[Image:IPLab1KidneyInfarction2.jpg|frameless]]
+File:IPLab1KidneyInfarction4.jpg|A higher-power photomicrograph shows the edge of this reddish area, illustrating coagulation necrosis (1) compared to the normal tissue (2). The necrotic tubules in this hemorrhagic, red infarct are hypereosinophilic. Compare the tubules on the right with the normal tubules seen in the left-hand portion of the slide. Note the interstitial hemorrhage which is associated with vascular leakage within this necrotic region in the tissue.
-| This is a closer view of the triangular-shaped infarction with the base at the cortical surface (1) and the apex at the corticomedullary junction (2).
+File:IPLab1KidneyInfarction5.jpg|This higher-power view of the infarct demonstrates retention of the tubular structure and cellular outlines. In the lower right-hand corner is a barely identifiable glomerulus (1). Note that, although the cellular architecture is retained, there are no nuclei within the renal tubular cells. The nuclei visible in this photomicrograph are the nuclei of inflammatory cells.
-|-
+File:IPLab1KidneyInfarction6.jpg|This section, taken at the cortical medullary junction, illustrates a blood vessel in the upper right portion of the slide (arrow) which is filled with thrombotic material. This vessel has occluded an end artery resulting in ischemia and infarction.
-| [[Image:IPLab1KidneyInfarction3.jpg|frameless]]
+File:IPLab1KidneyInfarction7.jpg|This gross photograph of an infarcted kidney is from another case. The triangular shape of an infarct is prominent on the right side of the image; the apex (arrow) of the triangle is evident at the corticomedullary junction.
-| This low-power photomicrograph of the kidney illustrates a sharply demarcated area of red discoloration extending from the capsule to the cortical medullary junction (arrow).
+</gallery>
 == Study Questions ==

← Older revision		Revision as of 03:12, 16 August 2013
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	{{Template:IPLab 1}}		{{Template:IPLab 1}}

−	[[Category:IPLab]]	+	[[Category:IPLab:Lab 1]]